The regulation of neurotransmitters DOES NOT include reuptake of the neurotransmission from the synaptic space degradation at the synaptic space O altering the rate of neurotransmitter release Ochanging the speed of receptor binding

The regulation of neurotransmitters DOES NOT include reuptake of the neurotransmission from the synaptic space degradation at the synaptic space O altering the rate of neurotransmitter release Ochanging the speed of receptor binding

Anatomy & Physiology

1st Edition

ISBN:9781938168130

Author:Kelly A. Young, James A. Wise, Peter DeSaix, Dean H. Kruse, Brandon Poe, Eddie Johnson, Jody E. Johnson, Oksana Korol, J. Gordon Betts, Mark Womble

Publisher:Kelly A. Young, James A. Wise, Peter DeSaix, Dean H. Kruse, Brandon Poe, Eddie Johnson, Jody E. Johnson, Oksana Korol, J. Gordon Betts, Mark Womble

Chapter12: The Nervous System And Nervous Tissue

Section: Chapter Questions

Problem 8ILQ: Watch this video (http://openstaxcollege.org/l/neurotrans) to learn about the release of a...

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Question

The regulation of neurotransmitters DOES NOT include
reuptake of the neurotransmission from the synaptic space
degradation at the synaptic space
O altering the rate of neurotransmitter release
Ochanging the speed of receptor binding

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Watch this video (http://openstaxcollege.org/l/neurotrans) to learn about the release of a neurotransmitter. The action potential reaches the end of the axon, called the axon terminal, and a chemical signal is released to tell the target cell to do something, either initiate a new action potential, or to suppress that activity. In a very short space, the electrical signal of the action potential is changed into the chemical signal of a neurotransmitter, and then back to electrical changes in the target cell membrane. What is the importance of voltage-gated calcium channels in the release of neurotransmitters?
The giant axon of the squid (Figure Q11–3) occu-pies a unique position in the history of our understandingof cell membrane potentials and nerve action. When anelectrode is stuck into an intact giant axon, the membranepotential registers –70 mV. When the axon, suspended in abath of seawater, is stimulated to conduct a nerve impulse,the membrane potential changes transiently from –70 mVto +40 mV.
If action potential propagation from one neuron to another neuron requires ACh release, what event at the synaptic cleft explains the need for action potentials to arrive at a high rate before the post-synaptic membrane achieves threshold? the lack of sodium voltage gated channels on the post-synaptic membrane O the rapid breakdown of ACh by ACHE at the synaptic cleft the slow release of ACh by the synaptic vessicles the slow movement of sodium through chemical gates Previous Next MacBook Air
Which of the following is true of ACh (acetylcholine)? O It cannot be degraded within the synaptic cleft to which it was released; it must be internalized and degraded by the cell that receives it O It is always excitatory because it causes the entrance of Na+ into a cell which leads to an increase in intracellular mV O It is released via exocytosis from the axon terminal of a somatic motor neuron O It is released via exocytosis from all pre-ganglionic motor neurons of the ANS
A loss-of-function mutation in the ion channel responsible for thegeneration of end-plate potentials results in a diminished permeabilityof the ion channel. 1) How would you experimentally test the effect of the mutation on thefunctional properties of the cell (synapse)? You should discuss the typesof postsynaptic responses and physiological parameters you couldmeasure.
The greater the influx of calcium into the presynaptic axon terminal, the greater the O magnitude of inhibition. release of neurotransmitter. O amplitude of the action potential O rate of reuptake by an enzyme
A muscle cell contains acetylcholine receptors that act as ligand-gated cation channels,and it also contains voltage-gated Na+ channels of the type found in neurons. Intheory, could a muscle cell get away with having only acetylcholine-gated cationchannels, which would serve the dual purpose of receiving neurotransmitter signalsand propagating action potentials? Why or why not?
Which statement is true regarding chemical neurotransmission? O An IPSP closer to the soma will diminish the effects of an EPSP on a dendrite Chemical neurotransmission is mediated by gap junctions All neurotransmitter receptors are located on the postsynaptic cell membrane Neurotransmitters remain in the synaptic cleft indefinitely
mbing fiber depolarizes mbrane potential to en Ca2+ channels. tage-gated channel 2+ Ca²+ Release 2+ Ca²+ Endoplasmic reticulum removal of AMPA receptors from the postsynaptic membrane. insertion of AMPA receptors into the postsynaptic membrane O increase in intracellular Ca2+ via voltage-gated Ca2+ channels O DAG- and Ca2+-mediated activation of PKC increase in intracellular Ca2+ via endoplasmic reticulum IP3 receptors
Excitatory neurons A and B both synapse with neuron C. Neuron A releasesa neurotransmitter, and neuron B releases the same type and amount ofneurotransmitter plus a neuromodulator that produces EPSPs in neuron C.Action potentials produced in neuron A alone can result in action potentialproduction in neuron C. Action potentials produced in neuron B alone alsocan cause action potential production in neuron C. Which results in moreaction potentials in neuron C, stimulation by only neuron A or stimulationby only neuron B? Explain.
An inhibitory postsynaptic potential (IPSP) is associated with O hyperpolarization a change in sodium ion permeability opening of voltage-regulated channels O the movement of calcium ions
Each of the following contributes to the unidirectional propagation of nerve signals EXCEPT_ A) presynaptic neurotransmitter secretion is separated from postsynaptic receptors. dendrites only receive signals. action potentials initiate the hillock and propagate only down the axon. D neuron membranes have a refractory period following generation of an action potential. E ion channels are oriented to transport ions only into the neuron.