One strategy for preventing type 1 hypersensitivity is to induce plasma cells to secrete lgG instead of IgE. Why would this block allergy symptoms
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One strategy for preventing type 1
hypersensitivity is to induce plasma cells to
secrete lgG instead of IgE. Why would this block
allergy symptoms?
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- a) How does Venetoclax, a BH3 mimetic, function in increasing cellular death? In your answer make sure you include what molecules Venetoclax binds and how this helps trigger cell death. b) How would addition of a BH3 mimetic increase cytoplasmic p53-mediated cellular death?T cells can differentiate into 5 different subsets of helper T cells. Answer the following questions for TH1 cells: A) In general, list three unique factors that differentiate the TH1 subset from the other subsets. B) For each of those factors identified in A, provide one specific example that applies to TH1 cells. C) What type of pathogens are targeted by the TH1 cells?c) State some of the features that cross the cell membrane and make "porin proteins" specific. d) Briefly clarify the concepts of "acylation", "prenylation" and "GPI stabilizer" in the context of membrane protein interactions. e) When determining ABO Blood Groups, give information about which blood group data can be obtained depending on the interaction of which antigens in the red blood cell and which antibodies in the serum.
- Poliovirus is highly cytopathic both in cultured cells and in animals. Inoculation of mice with poliovirus leads to a robust antibody and cellular response. Explain why mice infected with a poliovirus mutant that cannot cause cell death have poor antibody and cellular responses. What would be one strategy to restore robust antibody and cellular responses in mice infected with this mutant virus?How does thrombin catalyze the formation of loose fibrin and help in the activation of factor XIII?Why are CD8+/αβT-cells important? Explain the function of the mature activated CD8+/αβT-cell and list 3 cytotoxic effector molecules they secrete and 3 cytokines they secrete.
- IgM antibodies are much more efficient than IgG at activating the complement cascade. However, under certain circumstances, IgG antibodies will activate the complement pathway. One example of a situation in which IgG binding to its antigen will not trigger the complement cascade is when the IgG antibodies are neutralizing a bacterial toxin protein by blocking the receptor-attachment site on the toxin. the IgG antibodies are binding self-antigens, such as chromatin released from dead cells. the IgG antibodies bind to a bacterial surface by recognizing a repetitive polysaccharide component of the bacterial capsule. the IgG antibodies bind to a viral capsid protein that is present in more than 100 copies on the viral particle surface.Please explain the major regulatory checkpoints in necrosis signaling pathways?Give 5 storage conditions of human normal immunoglobulin injection and why?
- ''In the cellular regulatory pathways that controlcell growth and proliferation, the products of oncogenesare stimulatory components and the products of tumorsuppressor genes are inhibitory components.'' 'Is this statement true? Explain why or why not.It was stated that Rh antigens are so sparsely scattered in the red cell surface that IgG molecules bound to the Rh antigens are too far apart to fix C1q.Therefore, complement-mediated hemolysis cannot be invoked to explain hemolytic disease of the newborn. By what mechanism are the red cells destroyed?Because of oxygen and nutrient requirements, cells in a tissue must reside within 100 μm of a blood vessel. Based on this information, explain why many malignant tumors often possess gain-of-function mutations in one of the following genes: βFGF, TGF-α, and VEGF.