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- At normal to low blood glucose concentrations (1.e., roughly 110 mg/dl or less), the enzyme that is responsible for sequestering or trapping glucose within cells is which of the following enzymes? O hexokinase O glycogen synthase O glucose-6-phosphatase O glucokinaseWhich of the following statements about gluconeogenesis is correct? * Muscles have a large glycogen store which gives rise to blood glucose during prolonged starvation Fatty acids are plentiful in the blood during starvation and are used for glucose synthesis The enzyme glucose-6-phosphatase hydrolyses glucose-6-phosphate and is present in most cells. Gluconeogenesis enables the liver O to maintain blood glucose levels during starvationUnlike other sources of energy within the cell-like sugars- fatty acids such as triacylglycerols can be stored in anhydrous form. True O False
- The pentose phosphate pathway occurs in the mitochondrion of tissues actively engaged in synthesis of fatty acids. True O FalseThere must be a way to shut down glycogen breakdown quickly to prevent the wasteful depletion of glycogen after energy needs have been met. What mechanisms are employed to turn off glycogen breakdown?Glycogen synthase catalyzes glycogen synthesis. Determine whether each intermediate is associated with an “increase” or “decrease” and or “no effect” on the activity of glycogen synthase. Glucose 6-phosphate – cAMP –
- what effect would an increase in glucose typically have on the activity of glycogen synthaseGlycogen that is broken down in the muscle is not released as glucose into the circulation because the muscle lacks... Group of answer choices glucose-6-phosphatase phosphoryalse glycogen synthase hexokinaseAfter a carbohydrate-rich meal is consumed and the glucoserequirements of all tissues have been met, the liver begins tostore excess glucose in glycogen molecules. Explain the roleof the hexokinases in this phenomenon.
- What is the effect of a defective a(1→ 4) phosphatase in Pompe's disease (GSD II)? O Accumulation of abnormal, unbranched glycogen, leading to heart or liver failure and short fife expectancy O Glycogen accumulation in heart, leading to heart failure, muscle weakness, short life expectancy O Absence of UDP-glucose conversion to glycogen, leading to lack of muscle glycogen, exercise intolerance Glycogen accumulation due to lack of glucose-6-phosphate conversion to glucose, leading to liver enlargement, hypoglycemia, short life expectancy O Lack of complete degradation of glycogen to glucose. leading to mild hypoglycemia, enlarged liver and muscle weakness Submit Request AnswerFatty acids can be gluconeogenic precursors in plants, but not animals. O True FalseDuring ketosis brought on by a ketogenic diet, liver gluconeogenesis rates are high True False
