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List and briefly define/describe 9 different DNA –related aging mechanisms.
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- Genetic mutations can provide insights into the mechanisms of complex cellular or developmental processes. How might your analysis of a genetic mutation be different depending on whether a particular mutation is recessive or dominant?Explain the Role of telomere in regards to cancerexamine the process of gene expression. Include the following: Explain the regulation of gene expression in eukaryotic cells. Discuss mechanisms by which gene expression may be altered. How do these alterations induce cancer-causing mutations in cell DNA? Explain how cancer is formed. Describe genetic changes found in cancer cells and how these changes lead to alterations in cell behavior. Determine whether proteome data can be utilized in genetic disorder diagnosis. Relate the Human Genome Project data to the analysis of cancer genes. Relates the Human Genome Project's utility in pharmacogenomics and personalized medicine to diagnose and treat cancer
- Research cancer mutation. Provide the link to the research article that gives you your information. One good resource to use is PubMed. Then answer the following questions, in 3 paragraphs, 3-5 sentences each. 1. What kind of disease/cancer does this mutation cause? 2.What happens during transcription to cause this mutation? 3. Is this trait passed on to progeny? Can the progeny be a carrier or simply affected?Describe DNA Mutations: Definition: Define a mutation. Are they good or bad? What types of mutations can occur in the DNA? What can cause a mutation? Can mutation be “fixed”? What are the requirements for normal cell division? What are the requirements for cancer cells to divide and grow? Define the difference between a benign tumor, a malignant tumor, and metastatic cancer.Silent mutations that occur in DNA are quite common in living cells and usually involve no effects onphenotype. 3: Provide one example of a clinical implication of a “silent mutation” that proven to have an effect onthe phenotype and provide a brief description of its molecular characteristics?
- Mutagens, when introduced to the body can always cause cancer. Is this statement true? Select the correct response: No. Mutagens do not necessarily, directly equate to the cause of cancer. A cancer is usually caused by a substance called carcinogen that increases the probability of inducing a tumor. Mutagens can be limited to the result of spontaneous or induced mutations that causes changes and/or errors in the coding and reading of the DNA strand and in the genetic make- up of the organism involved. Though some mutagens are considered as carcinogens, not all mutagens can lead to cancer. Yes. The terms can be used interchangeably even in the medical field. Yes. Mutagens does equate to the cause of cancer. A cancer is usually caused by a substance called carcinogen that increases the probability of inducing a tumor. However, mutagens is not only limited to the result of spontaneous or induced mutations that causes changes and/or errors in the coding and reading of the DNA strand and in…(b): How many forms can naturally occurring DNA exist in? Explain how these forms are characterized. What properties of the functional groups determine the binding of DNA with anti-cancer agents? Explain.Essay: Relate the structure of the DNA to its role as a molecule for inheritance. Elaborate on the significance of DNA Describe the molecular mechanisms involved in P53’srole as a tumor repressor protein packaging in the inheritance and transmission of traits.
- (a) How does the duplication of individual genes occur? (b) individual that inherit a genetic condition known as xeroderma pigmentosum exhibit an extreme sensitivity to sunlight exposure and often develop skin cancer by the age of 10. What repair pathway is most likely disrupted as a result of such mutation? Explain why?Explain Telomere cell maintenanceI. The retinoic acid receptor (RAR) is a transcription factor that is similar to steroid hormone receptors. Thesubstance (ligand) that binds to this receptor is retinoicacid. One of the genes whose transcription is activatedby retinoic acid binding to the receptor is myoD. Thediagram that follows shows a schematic view of theRAR proteins produced by genes into which one oftwo different 12-base double-stranded oligonucleotides had been inserted in the ORF. The insertion site(a–m) associated with each mutant protein is indicatedwith the appropriate letter on the polypeptide map.For constructs encoding proteins a–e, oligonucleotide 1(5′ TTAATTAATTAA 3′ read off either strand) wasinserted into the RAR gene. For constructs encoding proteins f–m, oligonucleotide 2 (5′ CCGGCCGGCCGG 3′)was inserted into the gene.NH2 f g h i j k l m COOHa b c d eThe wild-type RAR protein can both bind DNA and activate transcription weakly in the absence of retinoic acid(RA) and strongly in RA’s presence. Each…