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- . Leukemia is a neoplastic (cancerous) proliferation of white blood cells. Clinicians are currently testing deoxycoformycin, an adenosine deaminase inhibitor, as a possible antileukemic agent. Why might one expect this therapy to be effective?a. When gene probes, fi ngerprinting, and sequencing make it possible for you to know about genetic diseases in you or one of your children, would you wish to use this technology to fi nd out? b. What if it were used as a screen for employment or insurance? c. Most of us would agree to growth hormone therapy for a child with dwarfi sm, but how do we deal with parents who want to give growth hormones to their 8-year-old son so that he will be “better at sports”?What is/are product(s) from commensal bacteria that regulate the expression for ler, regulator of genes in EHEC? a. Succinate b. Ethanolamine c. Fucose and sucrose d. None of these answers e. hydrogen thanks!!!!!!
- This is a blank question. Thank you in advance, Bloom Syndrome Bloom syndrome is a rare genetic disorder. It is characterized by short stature and a long narrow face with prominent nose and ears. There is also increased sensitivity to light. People who have the disorder often develop rashes on their face, forearms, and hands when they have been exposed to the sun. In addition, these people often suffer from chronic obstructive pulmonary disorder (COPD) and have a higher chance of developing cancer. The cause of this genetic disorder is a mutation in the BLM gene located on chromosome 15. The immediate effect of this mutation is that there is a defect in the functioning of the DNA helicase enzyme. What would be the effect of this mutation on DNA replication? What stage of the cell cycle would be most affected?2. Thiopurine methyltransferase (TPMT) metabolizes many immunosuppressants such as azathioprine. Genetic polymorphism changes the activity of this enzyme and thus the metabolic rate of transforming of drugs their corresponding metabolites. Define what the genetic polymorphism is. а. If a genetic variation leads to a reduction of TPMT activity, explain what outcomes will be expected to azathioprine. b.Our government has finite funds to devote to cancer research.Discuss which of the following areas of research you think shouldreceive the most funding.A. Identifying and characterizing oncogenes and tumorsuppressorgenesB. Identifying agents in our environment that cause cancerC. Identifying viruses that cause cancer D. Devising methods aimed at killing cancer cells in the bodyE. Informing the public of the risks involved in exposure tocarcinogensIn the long run, in which of these areas would you expect successfulresearch to be the most effective in decreasing human mortalitydue to cancer?
- ABOUT Phenylketonuria Explain Potential technical issues and limitations of PCR technology are mentioned Correct information about tissue that can be used to test for a genetic disease and justification of tissue selection Detailed information about the position (exact base pair number) of the new mutation relative to the sequence of the PAH gene. Numbering is based on the start of transcription of the PAH gene. PLEASE ANSWER ALLLL PLEASEEWhat is Prader–Willi syndrome? a. A genetic disorder characterized by loss of appetite b. Fat accumulation in the liver of leptin-resistant patients c. A failure to adapt to alternating periods of excess and inadequate energy intake d. A genetic disorder resulting in obesity e. Altered receptor activity for ghrelin. Mutations in an autosomal gene in humans cause aform of hemophilia called von Willebrand disease(vWD). This gene specifies a blood plasma proteincleverly called von Willebrand factor (vWF). vWFstabilizes factor VIII, a blood plasma protein specified by the wild-type hemophilia A gene. Factor VIIIis needed to form blood clots. Thus, factor VIII is rapidly destroyed in the absence of vWF.Which of the following might successfully be employed in the treatment of bleeding episodes in hemophiliac patients? Would the treatments workimmediately or only after some delay needed forprotein synthesis? Would the treatments have only ashort-term or a prolonged effect? Assume that allmutations are null (that is, the mutations result in thecomplete absence of the protein encoded by the gene)and that the plasma is cell-free.a. transfusion of plasma from normal blood into avWD patientb. transfusion of plasma from a vWD patient into adifferent vWD patientc. transfusion of plasma from a hemophilia A…
- T3 and T4a. require iodine for their production.b. are made from the amino acid tyrosine.c. are transported in the blood bound to thyroxine-bindingglobulin.d. All of these are correct.Why are fluoroquinolones only prescribed in severe cases? a. Phase 3 trial is still under progression b. Addiction can occur with severe withdrawal symptoms c. They are very expensive d. None e. Severe side effects and to prevent resistance developing Thank you!!!Q: Which of the following statements are true? I. Oncogenes isolated from a human tumor can transform mouse fibroblasts II. Mouse fibroblasts can be transformed by transfecting them with a single oncogene III. Human fibroblasts can be transformed by transfecting them with a single oncogene A. I and III B. III C. I, II and III D. II E. II and III F. I G. I and II