39. HIV could be used to cure Kaposi’s sarcoma by a. causing the cells to express exogenous surface cell markers b. activating genes for the expression of endogenous surface cell markers c. inactivating the patient’s tumor suppressing
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39. HIV could be used to cure Kaposi’s sarcoma by
a. causing the cells to express exogenous surface cell markers
b. activating genes for the expression of endogenous surface cell markers
c. inactivating the patient’s tumor suppressing genes
d. inactivating contact inhibition in the cell
e. inactivating cellular adherence
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- Which of the following statements are correct about inflammation and cancer (select all that apply)? A. Inflammatory cells are often detected in tumor samples B. The inflammatory cytokine TNF has been shown to have direct transforming properties C. Cancer often arises at sites of chronic inflammation D. Use of NSAIDs has been associated with increased mortality from colorectal cancer E. Levels of macrophage infiltration in tumors have been negatively correlated with patient prognosisADAM33 metalloproteinase inhibitors are sometimes co-administered with anti-tumor monoclonal antibodies in order to _____. a. block angiogenesis b. inhibit proteasome function in tumor cells c. enhance NK-cell killing by ADCC d. promote differentiation of dendritic cells e. reduce the surface expression of MHC class I on tumor cells.Which of the following drugs is a clinically-used monoclonal antibody for the treatment of breast cancer? O a. Doxycycline O b. Chloroquine O c. Herceptin O d. Protium
- 5) Briefly explain why the formation of a tumour can pose a risk to a person's homeostasis. 6) The functioning of the "Ras/MAPK" signal transduction pathway is absolutely essential in order for cells to grow, divide, and migrate. One important protein that is part of this pathway is BRAF. This protein is a kind of enzyme called a "kinase" – an enzyme that transfers a phosphate group onto another protein. In some melanomas, a mutated form of BRAF called BRAF Val600AGlu drives the progression of the cancer. The drug "vemurafenib" slows the progression of the cancer by slowing the production of the mutant BRAF protein. (National Cancer Institute. 2019. Types of Cancer Treatment. Retrieved from: https://www.cancer.gov/about-cancer/treatment/types/ Is this an example of a traditional cancer therapy or a targeted therapy? Briefly explain your reasoning in the space provided, using information provided in the text to support your answer. Type of therapy (traditional or targeted)?: Brief…1) A) List 15 drugs (monoclonal antibodies can be used) used clinically to treat cancer in humans. These targets must be signal transduction pathway components. B) For each drug, list the specific protein targeted. C) For each drug, describe the efficacy of treatment (i.e. what is the success rate in life extension) as well as appropriate cost of treatment whether it be per round or an average annual cost.Which of the following classes of oncogenes would likely be druggable with a small molecule (select all that apply)? A. Nuclear hormone receptor B. RAS C. Serine/Threoine Kinase D. Transcription Factor E. Receptor Tyrosine Kinase
- HIV could be used to cure Kaposi’s sarcoma bya. causing the cells to express exogenous surface cell markersb. activating genes for the expression of endogenous surface cell markersc. inactivating the patient’s tumor suppressing genesd. inactivating contact inhibition in the celle. inactivating cellular adherenceWhich ONE of the following is TRUE regarding adult T‐cell leukaemia‐lymphoma? Select one: A.The tumour cells have a CD4+ CD25+ phenotype B.It is usually cured with the CHOP chemotherapy regimen C.It is most commonly caused by infection with HIV D.It is commonly associated with hypocalcaemia at presentationWhich of the following statements are correct about the ability of cancer cells to form a secondary tumor at a site distant from the primary tumor (select all that apply)? A. Is possessed by majority of cells in a tumor B. Most micrometasases fail to become secondary tumor C. Usually occurs prior to establishment of micrometatstases D. Ability is acquired along with ability to move through circulation E. Is often acquired after micrometastases have been estabilished
- 4 Natalizumab is a therapeutic monoclonal antibody that targets the cell- adhesion molecule a4-integrin and is used as a second-line therapy for severe Crohn's disease. What might be its mechanism of action?Distinguish between What is known of CD105 (endoglin) as an hepatcellular carcinoma marker and it’s potential as a drug target. Discuss: - Is anything known about its biology? E.g does it have known ligands and normal functions? - if you were to target it would you just use it to get growth inhibiting compound into the new blood vessels or is CD105 required for neovascularization?Overexpression of Mdr1 in cancer cells causes the cells to be a. Apoptotic b. Susceptible to chemotherapy c. Resistant to chemotherapy d. Normal