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Why are leptin injections less helpful for most overweight people than for mice with the obese gene?
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- It is noticed that when a strain of mice exercises, their blood glucose drops to very low levels. Which of the following can explain this situation? a) These mice do not make enough insulin b) these mice lack insulin receptors on their cells c)these mice lack glucagon d) these mice cannot synthesize glycogen from glucoseAs a new, professional Laboratory Scientist, how do you think diabetes, regardless of kind or origin, is initially screened for or ruled out in clinical practice? Could you provide a list of common laboratory tests and explain why they are requested?What is the structural difference between insulin produced by the body and synthetic insulin given to diabetes patients? Provide images, description and explanation.
- 1)From Table 3, compute the mortality rate ratio for diabetes for Asian Americans compared to White America 2)Use the mortality rate ratio computed in question 1 to describe a health disparity in diabetes between Asian Americans and White Americans. 3)What information about diabetes mortality does Years of Potential Life Lost provide table 1 In your response, describe how this information is different from the information we get from diabetes mortality rate.5. There is an expression: diabetes mellitus is «hunger among abundance. What metabolic changes in diabetes confirm the validity of this statement? For answer: 9.8. Metabolic Changes in Diabetes Mellitus 535 a) list the main causes of metabolic changes in IDDM; b) enumerate the tissues with the main energy sources metabolism procceding according to this type of starvation; c) name the metabolic pathways that are activated and inhibited in these tissues, and explain why; d) list the symptoms of diabetes mellitus, that reflect such metabolic changes; c) draw a diagram of one of the mctabolic pathways that is activated in the liver under these conditions and explain the consequences of such activation.What are some practical implications of the finding that caloric restriction has long-term negative effects on body weight, cardiovascular disease, and late-life cognitive decline?
- Suppose that our test subject from problem 1 gained 55 pounds between the ages of 25 and 65 (a sadly common occur re nce) , and that her weight at 65 years of age is 175 pounds . Calculate how many excess calories she consumed per day to gain the 55 pounds over 40 years. Assume that our test subject is 5 feet 6 inches tall. What is her BMI? Would she be considered obese at 175 lbs ?Pancreatic beta cell dysfunction in type 2 diabetes is probably related in part to: a) Decreased advanced glycation end products b) Autoimmune destruction of the beta cells resulting in no insulin secretion c) Decreased beta cell mass d) Increased glucose-stimulated insulin secretionWhat is HbA1c? How does it differ from fasting blood glucose?
- For a cross-sectional study design that assesses the risk of developing type 2 diabetes among obese people - where would I collect my data from?A. What is the difference between preproinsulin and proinsulin? B. What is cleaved out of proinsulin to allow the mature insulin molecule to be formed? C. What is the C peptide and why is it medically significant? D. What is the purpose of the signal sequence and why isn’t it present in the mature insulin molecule?The hormones insulin and glucagon play an important role in the regulation of plasma glucose. b) People with Type II diabetes mellitus can manage their condition (maintain adequate glycaemic control) through use of oral hypoglycaemic drugs. Provide a rationale for how two hypoglycaemic drugs, with different mechanisms of action, can both work to lower blood glucose levels in Type II diabetes.
