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Explain the interactions between ADH, angiotensin-renin-aldosterone P, GFR, in a patient with acute hemorrhage.
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- there is a known cause of high blood pressure that occurs because of a reduced fluid flow that triggers RAAS and Na+ reaborption with increased blood volume and pressure. what is it?Define renin-angiotensin-aldosterone mechanismDescribe at least two mechanisms by which angiotensin II targets the kidneys to increase extracellular fluid volume and, therefore, increase blood pressure.
- Describe the response of the renin-angiotensinaldosterone hormone mechanism to a decrease in bloodpressure. How are extracellular fluid volume and urinevolume affected?Describe in detail the effects of angiotensin II.Renin is secreted in response to hypovolemia or to an increase in the osmolality of the blood. Provide three possible mechanisms by which these conditions may stimulate rennin secretion. What are the consequences of increased rennin secretion that lead to increased aldosterone secretion? Present this sequence of events. Why are angiotensin converting enzyme inhibitors (ACEIs) used as a common therapy for hypertension? How do they influence blood pressure? Are plasma levels of renin elevated or depressed in a patient with Conn’s disease? Why?
- Diagram/flow chart the reactions and interactions of the renin-angiotensin-aldosterone pathway. What conditiion is the primary stimulus for its activation? What would hapen to renin secretion in a patient with chronic kidney failure?ANP (atrial natriuretic peptide) can be considered to act against (in opposition to) the reninangiotensin-aldosterone-system (RAAS). What then does ANP try to achieve?List the four primary effects of angiotensin II.
- During treatment of congestive heart failure and hypertension, describe the mechanism of action and major side effects of loop diuretics, ACE inhibitors, blockers, and Ca2+ channel blockers.Contrast the effects of angiotensin II, aldosterone, and antidiuretic hormone on blood pressure with those of atrial natriuretic peptide.Explain the Summary of the renin-angiotensin system and the stimulation of aldosterone secretion by angiotensin II?