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Discuss the relationship between cancer and changes in gene expression that affect cell developmental processes.
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Solved in 3 steps
- Explain in detail two different genetic mutations that can promote the development of cancer. Include the name (or type) of gene mutated and whether the expression of the gene is upregulated or downregulated. Provide a description of the role the gene plays in regulating the cell cycle and how the cell is affected when the gene no longer produces a functional proteinDescribe two mechanisms by which genetic change causes cancer.Explain the molecular mechanisms of cancers caused by a P53 gene mutation.
- examine the process of gene expression. Include the following: Explain the regulation of gene expression in eukaryotic cells. Discuss mechanisms by which gene expression may be altered. How do these alterations induce cancer-causing mutations in cell DNA? Explain how cancer is formed. Describe genetic changes found in cancer cells and how these changes lead to alterations in cell behavior. Determine whether proteome data can be utilized in genetic disorder diagnosis. Relate the Human Genome Project data to the analysis of cancer genes. Relates the Human Genome Project's utility in pharmacogenomics and personalized medicine to diagnose and treat cancerExplain how p53 functions as a tumor suppressor gene. How can mutations in p53 lead to cancer, and how might gene therapy or other drug interventions inhibit the growth of a tumor?Explain how mutations in tumor-suppressor genes can be recessive at the level of the cell but cause dominantly inherited predispositions to cancer.
- Discuss why certain cell types are less likely and others are more likely to develop cancer than others.Describe the differences between point mutations, chromosomal translocations, and gene amplification in the process of cancer.Describe DNA Mutations: Definition: Define a mutation. Are they good or bad? What types of mutations can occur in the DNA? What can cause a mutation? Can mutation be “fixed”? What are the requirements for normal cell division? What are the requirements for cancer cells to divide and grow? Define the difference between a benign tumor, a malignant tumor, and metastatic cancer.
- Describe the steps by which the TP53 gene responds to DNA damage and/or cellular stress to promote cell-cycle arrest and apoptosis. Given that TP53 is a recessive gene and is not located on the X chromosome, why would people who inherit just one mutant copy of a recessive tumor-suppressor gene be at higher risk of developing cancer than those without the recessive gene?Discuss how oncogenes and tumor suppressor genes are related to genes involved in the control of normal growth and development.Compare and contrast oncogenes and tumor suppressors. Contrast oncogenes and proto-oncogenes. Describe the types of mutations that convert proto-oncogenes into oncogenes. Summarize some functions of common oncogenes in cell survival and uncontrolled growth. Contrast tumor suppressors to oncogenes. Describe the types of mutations in tumor suppressors that are found in common cancers. Summarize the functions of common tumor suppressors in cell survival and cell growth.