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Answer part D please
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- Equalizing the Expression of X Chromosome Genes in Males and Females Males have only one X chromosome and therefore only one copy of all genes on the X chromosome. Each gene is directly expressed, thus providing the basis of hemizygosity in males. Females have two X chromosomes, but one is always inactivated. Therefore, females, like males, have only one functional copy of all the genes on the X chromosome. Again, each gene must be directly expressed. Why, then, are females not considered hemizygous, and why are they not afflicted with sex-linked recessive diseases as often as males are?Mutation: Tan Body Pgeneration Phenotypes: Normal female X Tan male Fi generation Phenotvoe Females Males Total Ratio Normal Tan F, XF, Phenotypes: Normal female X Normal male F2 generation Phenotvoe Females Males Total Ratio Normal 8. 4. Tan 4 F Punnett square F. Punnett square Ths mutation is inhented aS 399F3 Other (mutant) Gene: Clot, Please use "cl" abbreviation C A homozygous wt organism has been crossed to a homozygous mutant for your gene and for withered wings (whd). The resulting heterozygote is test-crossed. Predict the test cross results.
- In rare instances, B cells can be found that have two immunoglobulin light chain alleles, both of which are rearranged in frame, and can encode functional light chain proteins. Yet, on the surface of the B cell, only one of the two light chain proteins is detected in the membrane-bound immunoglobulin receptor. The reason these rare cells have two functional light chain rearrangements but only express one of the two light chains as part of the B-cell receptor is: One of the two light chains is formed from rearrangement of a V gene segment that is a pseudogene. One of the two light chain proteins doesn’t form a stable complex with the heavy chain expressed in this cell. One of the two light chain alleles is not transcribed efficiently, and produces only low levels of protein. One of the two light chain alleles uses a V gene segment that is not targeted very often by the RAG recombinase. One of the two light chains is rapidly degraded after synthesis due to improper folding.This is a blank question. Thank you in advance, Bloom Syndrome Bloom syndrome is a rare genetic disorder. It is characterized by short stature and a long narrow face with prominent nose and ears. There is also increased sensitivity to light. People who have the disorder often develop rashes on their face, forearms, and hands when they have been exposed to the sun. In addition, these people often suffer from chronic obstructive pulmonary disorder (COPD) and have a higher chance of developing cancer. The cause of this genetic disorder is a mutation in the BLM gene located on chromosome 15. The immediate effect of this mutation is that there is a defect in the functioning of the DNA helicase enzyme. What would be the effect of this mutation on DNA replication? What stage of the cell cycle would be most affected?Retinoblastoma: The Hits Just Keep Coming Part IV – Time to Reconvene Question Since Julie is indeed a carrier of the mutated RB1 allele and Chris is homozygous for the wild-type allele, what is the likelihood that their next child will inherit Julie’s RB1 mutation? Part V – A Different Kind of Hit Question Chris does not have an RB1 mutation, and is therefore homozygous wild-type. Julie is heterozygous for the mutation. However, Kay has inherited a different RB1 mutation than the one her mother carries. Therefore, Kay did not receive her mother’s mutant allele. Assuming that Chris really is the father, what other explanation might there be for how she got a germline mutation?
- . Mutations in an autosomal gene in humans cause aform of hemophilia called von Willebrand disease(vWD). This gene specifies a blood plasma proteincleverly called von Willebrand factor (vWF). vWFstabilizes factor VIII, a blood plasma protein specified by the wild-type hemophilia A gene. Factor VIIIis needed to form blood clots. Thus, factor VIII is rapidly destroyed in the absence of vWF.Which of the following might successfully be employed in the treatment of bleeding episodes in hemophiliac patients? Would the treatments workimmediately or only after some delay needed forprotein synthesis? Would the treatments have only ashort-term or a prolonged effect? Assume that allmutations are null (that is, the mutations result in thecomplete absence of the protein encoded by the gene)and that the plasma is cell-free.a. transfusion of plasma from normal blood into avWD patientb. transfusion of plasma from a vWD patient into adifferent vWD patientc. transfusion of plasma from a hemophilia A…Equalizing the Expression of X Chromosome Genes in Males and Females Individuals with an XXY genotype are sterile males. If one X is inactivated early in embryogenesis, the genotype of the individual effectively becomes XY. Why will this individual not develop as a normal male?"The antibody diversity created by the combinatorial joining of V, D, and J segments by V(D)J recombination pales in comparison to the enormous diversity created by the random gain and loss of nucleotides at V, D, and J joining sites", is true or false.
- Epistasis Hypostatic gene Epistatic gene Dominant epistatic ratio Recessive epistatic ratio Lethal allele III III ||II A gene whose alleles are overridden or repressed. A gene that overrides or represses the effects of the alleles of another gene. How frequently a condition manifests in a population. Two genes interacting with one another to produce a novel outcome no matter what sort of inheritance patterns are found in either individual gene. The presence of an allele is fatal dependent on its dominant or recessive character. Reverses a prior mutation.HAIR COLOR 1. Select ANY allele to define your monster's hair color. It doesn't matter which one you select. Met His lle Gin Allele 1: TAC ATA CGC GGG ATT Arg Pro Ser Allele 2: TAC ATA CGC GTA ATT UCAG UA Allele 3: TAC GGG CCC AAA ATT Threonine Arginine Allele 4: TAC GGC CCT TTT ATT Lysine G ACU Leucine Submit Asparagine Aspartic 2 Leucine A U G acid Glutamic acid Phenylalanine AU G Alanine STOP CAGUGACUGAGU Cysteine Ser Gly STP Tyr Val O O O O Arg Glutamine Histidine Methionine Proline Isoleucine Arginine Serine Leu Phe Trp STP Cys Glycine Valine Asn Lys SerineTrue or false? Reasons that are acceptable by many for C-sections include all except triplets.