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- 8. In patients with diabetes mellitus type 1, the biochemical disorders result from changes in fuel metabolism. One of these signs is acidosis, Explain why such patients have a deviation of blood pH from the norm? For this 9. b) write the reactions of synthesis and oxidation of these molecules, name the enzymes, coenzymes, reaction localization: XAllosteric inhibitors of hemoglobin will decrease enzyme activity (oxygen binding) through which of the following mechanisms? Binding the enzyme in "T" conformation and displacing an activator Binding the enzyme and enhancing the Vmax Binding the enzyme, and keeping it in the "R" conformation Binding the enzyme, and keeping it in the "T" conformationOne of the following glycosaminoglycans is anti-coagulant O Heparan sulphate O Hyaluronic acid O Heparin sulphate O Keratan sulphate O Chondroitin sulfate search
- e/0/S Anemic patient diagnosed clinically through his symptoms which are: weakness, fatigue, shortness of breath, and dizziness. The lab. results of his hemoglobin level was less than 8 g/dl. On the examination of his Red blood cells isolated from the patient showed abnormally low level of lactic acid production. A deficiency of which one of the following enzymes would be the most likely cause of this patient's anemia? * Pyruvate kinase Hexokinase All of the above Phosphofructokinase Phosphoglucose isomerase Lactate dehydrogenaseCompare and contrast the c4 pathway and the malate–aspartate shuttle in bullet format, meaning list the similarities and differences.The following structure is more active than morphine as an analgesic. H3C CH3 H. H3C' O Il.. What is the principal reason for this? The extra acetyl group binds to cell surface receptors improving active internalization. The acetyl groups mask a polar group allowing the structure to cross the blood brain barrier more efficiently. The extra acetyl group masks an alcohol group which would otherwise undergo a phase Il metabolic conjugation reaction leading to rapid excretion of the drug. The acetyl groups increase the stability of the drug in the cellular environment. O Il..
- Describe the mechanism of toxicity of P=S organophosphates insecticides (e. g. parathion) in the body of an insectA schematic representation of the enzyme IspD complexed to inhibitor 3, and a series of inhibitors 3-5 are shown below. Ala202 lle240 mwww NH NH Val263 ОН www HN N- lle177 HN 'N' CI 3 X = N 4 X = C-CN 5 X = C-COO IC50 274 µM IC50 140 nM IC50 35 nM NH2 HN Val266 N -N O-H---- N HN %3D Arg157 HN wwww lle265 Explain why structure 4 is a more potent inhibitor (lower IC50 value) than inhibitor 3 and why structure 5 is a much weaker inhibitor (higher IC50 value) than 3 and 4.I. Predict the outcome of the following reactions. If positive write what will you observe (color, name of the product). If negative, explain the reason why. NH Orcinol но NH2 A 1.) ÓH H. Но NH 2.) ОН ОН HO-P-O-P-0-P- OH (NH4)ẠM0O4 ÓH OH ÓH Conc. HNO3 3.) Он H Conc. HNO3 HN -NH N. КОН 4.) H3C NH HO Brz in H20 Ba(OH)2 HO- 5.) ÓH
- create or design a concept map explaining in one page using figures, illustrations, and a few descriptions or explanations of the Type 1 glycogen storage disease. The content of your concept map should include the biochemical and molecular basis of the disease with clinical presentation and therapeutic options.Which of these heterocyclic drugs is likely to be the least soluble in water? Use the Fsp³ parameter to decide. OH Tramadol Chemical Formula: C16H25NO2 YOUR OW Pantoprazole Torasemide Chemical Formula: C16H15F2N3O4S Chemical Formula: C16H20N4O3S Temazepam -OH Chemical Formula: C16H13CIN₂O2 Tioconazole Chemical Formula: C16H13C3N₂OS A. Tramadol B. Pantoprazole C. Torasemide D. Temazepam E. ToconazoleI. Predict the outcome of the following reactions. If positive write what will you observe (color, name of the product). If negative, explain the reason why. NH Orcinol но- NH2 A 1.) OH 'NH но NH A 2.) ОН ОН HO-P-O-P-O он (NH4)4MOO4 OH OH OH Conc. HNO; 3.) OH