Degradation of normal glycogen results to 97.2% glucose 1-phosphate and 7.2% glucose. Assuming a ratio of glucose 1-phosphate to glucose recorded in a glycogen sample from a patient with a liver disease was 100, The probable structure of the patient’s glycogen is________
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Degradation of normal glycogen results to 97.2% glucose 1-phosphate and 7.2% glucose.
Assuming a ratio of glucose 1-phosphate to glucose recorded in a glycogen sample from a patient with a liver disease was 100,
The probable structure of the patient’s glycogen is________
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- A mature glycogen particle extends out from the homodimer, glycogenin, typically having 12 tiers of chains with two chains per tier and 13 residues per chain. Calculate the total number of chains from the 1st to the 7th tier of such a glycogen molecule. _________In 2-3 paragraphs (must be typed) explain: Discuss the regulation of cholesterol synthesis.Which glycogen storage disorder is known as amylopectinosis? explain very briefly.
- The cleavage of fructose-1,6-bisphosphate to glyceraldehyde3-phosphate and dihydroxyacetone phosphate is an exampleof an __________________ reactionPyridoxal phosphate (PLP) has the main function of converting one type of amino acid to another amino acid via _______________________. transanimation carboxylation hydroxylation cis-animationBased on this video https://www.youtube.com/watch?v=rKng5-ij6kQ Provide a schematic diagram for the Fehling’s test methodologies in determining the presence of carbohydrates. Also, give the basic principle for the test. (not a graded question, video is very brief)
- TRUE OR FALSE (Glucose), Fructose and Sucrose gave a positive result to Seliwanoff’s Test Beeswax when dehydrated by KHSO4 gave a (pungent odor) when heated.Dysregulation of glutamate dehydrogenase (GDH), an enzyme that catalyzes the oxidative deamination of glutamate, leads to a variety of metabolic and neurological disorders. One such disorder is hyperinsulinism/hyperammonemia syndrome. 1. Describe the role of GDH including the structure, function and biochemistry of the protein and its contribution to the disease. 2. Discuss the several ways that this disease can be diagnosed in the lab 3. Discuss the mechanism by which pharmacological agents act to help in the management of the disease.The “keto diet” is a dietary regimen in which carbohydrates are severely restricted, protein intake is low, and fat comprises 70–90% of daily calories. The diet causes “nutritional ketosis,” which is a different metabolic state than the ketosis that occurs in an untreated diabetic. The normal ketone body concentration is less than 0.6 mM. Explain why an individual on the keto diet may have a blood ketone body concentration of up to 3 mM. What biochemical pathways are active in an individual adhering to the keto diet? What pathways are largely inactive? Protein intake is kept low because a high-protein diet would inhibit ketosis. Explain why. Why might the keto diet lead to weight loss?
- The rate of the reaction of glycogen N with inorganic phosphate, Pi, to form glucose-1-phosphate and glycogen N-1 is:The following laboratory test results were obtained in a patient with severe jaundice, right upper quadrant abdominal pain, fever, and chills. Serum alkaline phosphatase Four times normalSerum cholesterol IncreasedAST (SGOT) Normal or slightly increased5’-Nucleotidase IncreasedTotal serum bilirubin 25 mg/dLConjugated bilirubin 19 mg/dLProthrombin time Prolonged but improves with a vitamin K injection A) What is the most likely cause of jaundice in the patient? Rationalize your answers.The major enzymatic defense against oxidative stress is provided by superoxide dismutase, glutathione, peroxidase, peroxiredoxin, and _____________